Physical Characteristics of patient with Chronic Heart Failure (Term Paper Sample)

Chronic Heart Failure
Name
Institutional Affiliation
Chronic Heart Failure
Brown is a 53-year-old male diagnosed with chronic heart failure in the past five months. Initially, during his youthful days in high school and college, Brown was actively engaged in physical exercises and sports activities, which kept him physically fit and healthy. After graduating from college, a global technology corporation employed Brown as a program developer, where he has worked for the past 23 years. His job as a program developer did not require him to move around physically. Further, Brown drives from home to workplace, and again from workplace to home. After Brown realized he was suffering from chronic heart failure, he came up with a plan to lead a healthy lifestyle. In the past few weeks, Brown engaged in physical activities. Once every week, Brown engages in resistance training sessions and jogging in his neighbourhood, each event taking approximately thirty minutes. However, Brown has not yet noted his dream physical fitness and healthy body coming back and focuses on drawing an exercise program that will aid in improving his health at a fast rate.
Physical Characteristics
Height: 5’8”
Weight: 199 lbs
Heart rate: 72 beats/min
Blood pressure: 138/86 mmHg
BMI: 30.3
Body fat: 32.1%
Waist circumference: 42 in.
Blood Lipid Profile
Fasting glucose: 70 mg/dl
HbA1c: 6.2%
GXT
Maximum BP: 189/97 mmHg
Maximum HR: 174 beats/min
VO2max:: 27.5 ml/kg/min
Random blood glucose before test: 164 mg/dl
Pathophysiology
Heart failure has evolved to be one of the common diseases globally, affecting many individuals. In developed nations, chronic heart failure is turning out prevalent, especially among the ageing population. Chronic heart failure is progressive ineffectiveness of the heart to carry out its role of pumping sufficient blood in the whole body to make sure enough oxygen supply (Adebayo, Olunuga, Durodola, & Ogah, 2017). Chronic heart failure thrives as a central reason behind the admission of many individuals above the age of 65 years of age, as well as one of the costly disease categories (Adebayo, Olunuga, Durodola, & Ogah, 2017). The readmission rate to healthcare facilities after discharge also thrives high for heart failure compared to other complicated diseases.
Heart failure has evolved as a clinical syndrome, making it to have numerous aetiologies. In the western sphere, the common underlying chronic heart failure causes comprise hypertension, ischaemic heart disease, and idiopathic dilated cardiomyopathy (Wright & Thomas, 2018). For the case of the developing nations, some core heart failure causes include rheumatic heart disease, valvular illness, and nutritional deficiencies (Wright & Thomas, 2018). However, among all these causes, hypertension remains the core cause of heart failure, although many people underestimate it. Chromic hearth failure is highly dependent on distinct symptoms consistent with exertional dyspnoea and excessive fatigue, which depends on a patient’s fitness level, age, and reduction in cardiac function.
The major symptoms among patients suffering from chronic heart failure comprise dyspnoea and muscle fatigue. Dyspnoea is common chronic heart failure, whereby its changes are traceable in the lungs (Wright & Thomas, 2018). Patients suffering from chronic heart failure experience an excessive fatigability of respiratory musculature. In some cases, the lung structure can be abnormal among chronic heart failure patients due to the irreversible and pulmonary arterial fluctuations that might develop. On the other hand, muscle fatigue can come along due to the combination of a decrease in blood flow to the active skeletal muscle and muscle tissue loss in the margin. Excessive vasoconstriction of muscle vasculature and endothelial dysfunction appear common with chronic heart failure syndrome (Katz, 2017). Among patients with chronic heart failure, skeletal muscle emerges to be abnormal in diverse ways. For instance, a patient might experience reduced peak strength and early fatigability of heart failure muscle. In such a case, the patient might frequently complain of muscle fatigue as a core limitation while performing their daily chores.
Metabolism of skeletal muscle during exercise thrives as a profound abnormality among patients, whether chromic hear failure is merging from dilated cardiomyopathy or secondary to an ischaemic heart complication. In such cases, a patient might experience new phosphocreatine bonds depletion and early acidification upon engagement to a physical exercise (Katz, 2017). Even though chronically impaired blood flow might emerge as a contributor to the muscle